G-CSF treatment decreased the gene expression of triglyceride (SREBP1c, SCD-1, FAS), cholesterol (SREBP2, HMG-CoA reductase) biosynthesis and inflammation markers (TNF-α, MCP-1), in all G1, G2 and G3 groups as compared to the HF group (Figure 3A). The gene discussed is SREBF1; the disease is hydrops fetalis.