who incubated primary ovarian cancer cells with 5-aza-2′-deoxycytidine, a deoxy derivative of 5-Aza, which resulted in the hypomethylation of an STAT-1 binding site upstream of the promoter region and an exacerbated release of CXCL10 following IFN-γ stimulation (Peng et al., 2015). This evidence concerns the gene STAT1 and ovarian carcinoma.