Active AKT is known to phosphorylate FoxO3 at Thr32 and Ser253, as demonstrated here for both ex vivo‐isolated human IPF fibroblasts and for control human lung fibroblasts exposed to pro‐proliferative growth factors in vitro (Brunet et al, 1999; Dobson et al, 2011). Here, FOXO3 is linked to idiopathic pulmonary fibrosis.