DOCK5 and infection: Without influenza infection (i.e., MOCK infection), DOCK5 predominantly modulates cell migration, as indicated by the significant enrichment of cell receptor signaling (FET p = 1.3e−8, 2.4-fold), cell communication (4.9e−4, 1.3-fold), cell migration (FET p = 6.1e−3, 1.9-fold), extracellular matrix receptor interaction (FET p = 8.6e−3, 2.8-fold), and integrin pathways (FET p = 2.5e−2, 3.8-fold) in the DEG signature in the MOCK DOCK5-ko cells in comparison with the MOCK wildtype cells (Tables S14 and S15).