To examine the relevance of these findings to human TB infection, we investigated IL‐17, IL‐22, and IL‐23 concentrations in BALF from 17 well‐characterized patients with confirmed pulmonary TB and 18 well‐matched respiratory symptomatic patients with other diagnoses that may cause a clinically similar picture, which we have previously reported 44. This evidence concerns the gene IL17A and pulmonary tuberculosis.