To investigate whether this in vitro effect occurred in vivo, we analyzed the presence of infiltrating monocytes and CD73 expression in a swine model of MI locally treated with porcine cATMSCs, previously reported by our group to reduce the amount of infiltrating effector T cells in the infarcted tissue and to ameliorate the regeneration of the myocardium (15, 16). The gene discussed is NT5E; the disease is myocardial infarction.