In DH skin lesions, higher expression of STAT3 and STAT4 was observed, which may contribute to crucial role of IL-6, TNF-α, IL-8, IL-17, and IL-12 in pathogenesis of DH, which is consistent with findings concerning cytokine levels in DH [65–67]. This evidence concerns the gene STAT3 and dermatitis herpetiformis, familial.