TARDBP and amyotrophic lateral sclerosis: The strong enrichment of TDP‐43 interactors and other causal genes for ALS within M2 and M6 modules further reinforces their association with the ALS/FTD spectrum and suggests that proteins within these modules have critical roles in mechanisms that drive TDP‐43 aggregation and other cellular‐driven pathological processes (i.e., neuroinflammation).