KRAS and cancer: This is further supported by higher basal p100 Prdx1 levels (Figure 3A), high MW p28 Srxn (Supplementary Figure 12) levels, and differential CSA responses (Figure 3C) in Panc1 and ASPC-1 cancer cells as compared to normal ductal HPDE cells and Kras mutant HPDE-Kras cells.