EML4 and cancer: Oligomerization and activation of ALK due to distinct intracellular localization at microtubules (EML4, KIF5B, TFG, DCTN1, PTPN3, and KLC1) or cellular membranes (SQSTM1, TPR, CRIM1, STRN, HIP1, and CLTC) is likely to be of central relevance for ALK-mediated oncogenesis and presents a new therapeutic target for the treatment of ALK fusion driven cancers.