In oocytes, levels of two rapidly-degraded anti-apoptotic proteins, cellular Fas-associating protein with death domain-like interleukin-1-converting enzyme-like inhibitory protein long form (c-FLIPL) and XIAP, were decreased in wild-type cells treated in such a way as to activate eEF2K (doxorubicin, an anti-cancer agent [46]). The gene discussed is EEF2K; the disease is cancer.