Since the 20% remaining expression of Arg1 in the lungs of OVA/OVA-treated Arg1-KOLysM mediated an intermediate phenotype between OVA/OVA-treated Arg1-Con and Arg1-KOTie2Cre mice, we limited our description of the effects of pulmonary Arg1 deficiency on allergic asthma to Arg1-KOTie2Cre mice. Here, ARG1 is linked to allergic asthma.