Helminth‐induced IL‐10 may drive immunoglobulin class switching to IgG413, 14 which, akin to the Th2 cytokine‐induced15 polyclonally stimulated IgE, may inhibit development of allergen‐specific effector responses,5, 16 leading to inverse helminth‐allergy associations. Here, IL10 is linked to allergic disease.