CD36 and myocardial infarction: Within hours after MI, uptake of apoptotic and necrotic cardiomyocyte debris was mediated by CD36 on Ly6Chi monocytes and the importance of CD36-mediated clearance by Ly6Chi monocytes was revealed in CD36-deficient bone marrow recipients, which displayed increased infarct size early after MI compared to WT recipients (63).