PRL and peripartum cardiomyopathy: In regard to the PRL/vasoinhibin axis, the shared hallmark of these diseases is the dysregulation of vasoinhibin levels, that means they may either be too low to execute a physiological function—such as the suppression of angiogenesis in the eye—or too high, which may lead to detrimental effects on the microvasculature of the heart, as proposed for the etiopathology of peripartum cardiomyopathy (PPCM).