Based on our finding demonstrating that transfection of miR-29b mimics significantly downregulates directly or indirectly a variety of inflammatory cytokines and chemokines at mRNA and/or protein level, we investigated the putative role of the known receptors of these molecules (CCR1, CCR2, CCR3, CCR4, CCR5, CXCR1, CXCR2, CXCR3, CXCR6, IL10RA, IL10RB, IL12RB1, IL17RA and IL23R) in MM disease pathobiology by performing an in silico analysis on MM gene expression data sets. The gene discussed is CCR2; the disease is Miyoshi myopathy.