EGFR and lung cancer: Furthermore, we found that induction of miR-370 over-expression attenuated the EGFR expression and inhibited the proliferation, clonogenicity, and invasion of lung cancer cells while inhibition of endogenous miR-370 by transfection with miR-370 inhibitor significantly enhanced the EGFR expression and the proliferation, clonogenicity, and invasion of lung cancer cells.