Myxinidin2 and myxinidin3 inhibited the inflammatory response to infection with S. aureus CCARM 3018, A. baumannii 719705, and P. aeruginosa 4076 (4 × 107 colony-forming units (CFU)/mL) by suppressing the phosphorylation (activation) of STAT3, JNK, and p38 (Figure 8A-8D). The gene discussed is STAT3; the disease is infection.