Type 1 diabetes, also known as insulin‐dependent or juvenile diabetes, results from a deficiency of insulin due to the T‐cell‐mediated destruction of its secretion source, β‐cells.5 Type 2 diabetes involves the inability of cells in the liver, muscles, and adipose tissue to respond to the normal functions of insulin.6, 7 Patients with type 1 diabetes are characterized by hyperglycemia and hypoinsulinemia, generally without insulin resistance. This evidence concerns the gene INS and type 2 diabetes mellitus.