NFKB1 and posterior cortical atrophy: GSEA showed that a negatively enriched expression of genes sets was involved in hallmarks of transforming growth factor beta (TGF-β) signaling (NES = −1.79, FDR = 0.01, P = 0.015) (Fig. 3h), tumor necrosis factor alpha (TNF-α) via nuclear factor kappa-light-chain-enhancer of activated B cells (NF-kB) signaling (NES = −1.77, FDR = 0.005, P = 0.017) (Fig. 3i) and KRAS prostate up signaling (NES = −1.69, FDR = 0.08, P = 0.203) (Fig. 3j) in miR-193a-overexpressing PCa cells.