GNRH1 and ovarian hyperstimulation syndrome: Taskin et al. [28] detected significantly lower ovarian weight, estrogen levels and corpus luteum counts with lower IL-6 intensity and VEGF expression in GnRH antagonist treated animals than in OHSS animals and attributed development of OHSS to increased expression of VEGF secondary to increased IL-6 intensity; thus raising a possibility of inflammatory basis for development of OHSS.