We also observed the downregulation of the protein kinases ATM (ataxia-telangiectasia mutated) (logFC = −2.46, p-value < 0.0001 and FDR < 0.00001), an activator of the DNA damage response in the face of DNA double strand breaks (DSBs), and NLK (nemo-like kinase) (logFC = −2.55, p-value < 0.0001 and FDR < 0.0001), a p53 activator, in eBL tumor cells. This evidence concerns the gene ATM and neoplasm.