Friedreich ataxia (FRDA), the most common autosomal recessive hereditary ataxia, is majorly caused by homozygous expanded guanine-adenine-adenine (GAA) repeats in intron 1 of the frataxin (FXN) gene (Campuzano et al., 1996; Lynch et al., 2012; Lynch and Seyer, 2014). The gene discussed is FXN; the disease is Friedreich ataxia.