Taken together, our findings of reduced hepatic fibrosis area, reduced number of activated HSC and macrophages, reduced levels of TGF-β1 and polarization to M2 macrophages are consistent with those seen in our previous study, when we administered hAEC alone in the CCl4 mouse model (Manuelpillai et al., 2012). Here, TGFB1 is linked to Hepatic fibrosis.