CSF2 and atherosclerosis: Mice that lack TRα have higher levels of circulating pro-inflammatory cytokines at baseline (26), excessive secretion of pro-inflammatory cytokines by unstimulated macrophages (26, 27), a lower induction of the pro-inflammatory cytokine granulocyte-macrophage colony-stimulating factor (GM-CSF) during acute inflammation (5) and impaired macrophage function in an atherosclerosis model (26).