TNFSF13B and Miyoshi myopathy: Our study demonstrated that MΦs had increased expression of BAFF compared with monocytes, and the secretion level of BAFF in MΦs was higher than that in MM cell lines, which was in accordance with previous reports that BAFF signaling acted mainly through a paracrine system rather than an autocrine mechanism.13, 17 We also demonstrated that PBMC-induced MΦs could protect MM cells from both spontaneous and bort-induced apoptosis.