These pathways include but are not limited to P53 signaling which can be triggered due to different stress signal and may result in cell cycle arrest and apoptosis, Nod-like receptor signaling which leads to the activation of various proinflammatory cytokines such as IL-18 and IL1-β, after the activation of various inflammasomes, Hypertrophic cardiomyopathy, dilated cardiomyopathy, Toll-like receptor signaling pathway, T-cell receptor signaling pathway and Renin-angiotensin system pathway. This evidence concerns the gene TP53 and dilated cardiomyopathy.