AKT1 and glioblastoma: Based on the results of our previous publication [22], we observed specific inhibition of AKT1 phosphorylation by LY294002 (50 μM); a downregulation of NF-κB p65-Ser536 phosphorylation only by combined treatment of BMS344551 (20 μM) and LY294002 (50 μM); specific inhibition of ERK1/2 phosphorylation by U0126 (10 μM) in both glioblastoma lines (Figure 5A).