HRAS and cancer: Because a previous study demonstrated that SV40LT enabled conversion of some types of normal cells into cancerous cells [20], we examined whether SV40LT-overexpressing K9 fetus 1 cells showed cancer cell properties by comparison with SV40LT-overexpressing K9 fetus 1 cells transduced with H-RASV12, an oncogenic mutant of H-RAS (substitution of the 12th glycine to valine) (Fig. 2a).