Other studies demonstrated further data supporting a thesis that some pathological phenomena such as leukotriene pathways [31], circulating platelet-leukocyte aggregates [32], activation of toll-like receptors (TLRs, [33]), release of platelet activating factor (PAF) from mast cells [34] or infection with Chlamydia pneumoniae [6] are common for asthma and atherosclerosis. This evidence concerns the gene PCLAF and atherosclerosis.