This process is accompanied by an increase in pulmonary fibrosis markers, such as α-SMA, collagen, vimentin, and Snail, and has also gained considerable research attention in the past few years as a potential contributor to pulmonary fibrosis.38, 39, 40 Our data showed that lnc-PCF could stimulate the proliferation of myofibroblast derived from epithelial transition by facilitating the driving cell cycle progression. The gene discussed is SNAI1; the disease is pulmonary fibrosis.