NGP17B (Galα(1,3)Galα-BSA), NGP12B (Galα(1,4)Galβ-BSA), and NGP5B (Galα(1,6)Galβ-BSA) showed the best differential reactivity between active CL vs. cured CL infection, and active CL vs. heterologous infection. This evidence concerns the gene NAGA and infection.