Infrequently, severe hepatic dysfunction presenting as fulminant hepatic failure is also reported in dengue infection.5-8 About 65-97% of dengue patients have derangement of liver biomarker aspartate aminotransferase (AST) and alanine aminotransferase, with maximum rise during the convalescence (days 7–10).9,10 In areas where dengue is common, it is considered as one of the important causes of acute viral hepatitis.11 Liver enzyme derangement is associated with prolonged stay in hospital, development and increased severity of complications and is considered a poor prognostic marker.12,13. The gene discussed is GPT; the disease is dengue disease.