The proposed mechanism by which GTE produces beneficialeffects on serum lipid profile is due to its EpigallocatechinGallate(EGCG) componentwhichforms complexes with lipids and lipolytic enzymes, thereby interfering the process of luminal emulsification, hydrolysis, micellarsolubilization and subsequent uptake of lipids.22 Research also suggests linalool and EGCG improves lipid profile and obesity by acting on peroxisome proliferator activated receptor (PPAR-α) which is the site of action of standard lipid lowering agent gemfibrozil.23 Here, PPARA is linked to obesity due to melanocortin 4 receptor deficiency.