Since at the same time NFATc1, a HF quiescence marker, occurring upstream of Bmp signaling was upregulated and a downregulation of some Bmp antagonists (e.g. BAMBI) is seen, we assume that the delicately balanced Bmp pathway, involving an intimate interaction of many genes and cellular events is nevertheless differentially regulated thus resulting in telogen arrest despite the downregulation of some BMP signaling molecules [44–46]. The gene discussed is BAMBI; the disease is hydrops fetalis.