The results presented by Silva and Jones65 suggested that the expression and production of the BHV-1-infected cell protein 0 (bICP0) is interfered with by Bovine Herpes Virus1 (BHV-1) miRNAs, which are expressed during latent infection and stimulated the RIG-I signalling pathway, which correlated with activated type I interferon signalling. The gene discussed is RIGI; the disease is disease arising from reactivation of latent virus.