The myoclonic phenotype might resemble FBDS in human autoimmune patients.44 However, unlike patients with LGI1 antibody encephalitis, FBDS are not reported in patients with human mutations of the LGI1 gene, which causes autosomal dominant lateral temporal lobe epilepsy with auditory hallucinations.45 Nonetheless, some interesting similarities in seizure semiology do exist between the groups. The gene discussed is LGI1; the disease is autosomal dominant epilepsy with auditory features.