MTOR and infection: The fact that all functions associated with miR-125a are shared by miR-30e-5p, allowed us to hypothesize about the relevant roles for pathways specifically regulated by overexpressing miR-30e-5p early after infection with virulent E75 ASFV strain, and contributing to the inhibition of key macrophage innate defensive mechanisms such as mTOR, toll-like receptors, RIG-I-like receptors, cell adhesion molecules, chemokine signaling pathways, Fc gamma R-mediated phagocytosis, leukocyte transendothelial migration or CAMs.