Further experiments validated the targeting effect of miR‐1 on Src and the following cancer‐related functions of miR‐1 via Src in esophageal cancer cell line, which is concordant with the previous study that overexpression of miR‐1 reduces cell viability in HepG2 cells 32 and delays the growth rate of breast cancer stem cells, gastric and colorectal cancers 33, 34. This evidence concerns the gene SRC and breast cancer.