LGALS4 and oculocerebrorenal syndrome: To test how the dOCRL phosphatase activity contributes to its role in hemocytes, we performed rescue experiments in the docrl mutant using He-Gal4-mediated expression of wild-type dOCRL, a phosphatase-inactive dOCRL (dOCRLH469R, corresponding to a Lowe Syndrome mutation [41]), or the dOCRL phosphatase domain alone [42].