Accordingly, a crucial contribution of MFN2 to the effect of RSV was demonstrated by highly branched mitochondrial networks in C2C12 myoblasts, in the prostate cancer cell line, P3, and in mouse embryonic fibroblasts (MEFs) after treatment with 10–20 μM RSV for 48 h, due to stimulated MFN2 expression [59]. This evidence concerns the gene MFN2 and prostate carcinoma.