In view of the facts that netrin-1 and DCC have also been found to be involved in these plastic processes [23], and that the overexpression of netrin-1 improves the functional recovery and reduces the infarct size in animal stroke models [42, 43], we measured the temporal and spatial expression patterns of netrin-1 and DCC in rat brains after ADSC transplantation. The gene discussed is DCC; the disease is Stroke.