Therefore, ascertaining that oxidative stress and inflammatory process are driving force in AD pathology and that Nrf2 activators and anti‐NF‐κB strategies are considered as network medicines in multiple neurodegenerative diseases and surely efficacious AD treatment strategy 28, in this study we evaluated the role of DMF and MMF, through NF‐κB‐Nrf2 signalling pathways, in an in vitro model of AD in SH‐SY5Y cells. This evidence concerns the gene NFKB1 and Alzheimer disease.