Furthermore, inhibition of autophagy by Baf-A1 challenge resulted in further accumulation of both MAP1LC3B-II and SQSTM1 in AGS cells after 6 h of Hp-WT or Hp-ΔcagA infection (Figure 3F), suggesting that H. pylori CagA did not inhibit the fusion of autophagosomes with lysosomes. The gene discussed is HP; the disease is infection.