And more recently, Ben-Ami et al. showed that drug-resistance of Aspergilus fumigatus in common pulmonary infections of cancer patients was attributed to its ability to cause occlusion in pulmonary vasculatures via secretion of anti-angiogenic metabolites [13], and later showed that co-administration of pro-angiogenic factors such as basic fibroblast growth factor (bFGF) and vascular endothelial growth factor (VEGF) significantly amplified the effectiveness of antifungal treatments by increasing the accumulation of monocytes in the infected tissues [14]. This evidence concerns the gene FGF2 and cancer.