Since SET Isoform 2 was more consistently overexpressed in pancreatic cancers (Figure 1) and SET Isoform-2 induced more prominent EMT-related phenotypic changes in PANC-1 as compared with SET Isoform 1 (Figure 3 & Supplementary Figure 4), we focused only on SET Isoform 2 for further mechanistic studies. The gene discussed is SET; the disease is pancreatic neoplasm.