Another factor that could have stimulated satiety was the presence of phloridzin in the meal, which has anti-obesity effects related to suppression of sodium-dependent glucose transporter-1 (SGLT1) [16] and to the inhibitory effects of phloretin, the aglycone of phloridzin, on α-glucosidase activity [41]. The gene discussed is SLC5A1; the disease is Obesity.