ITGAX and experimental autoimmune encephalomyelitis: We have previously shown in an animal model for multiple sclerosis, experimental autoimmune encephalomyelitis (EAE), that while CD11c+ microglia are effective antigen‐presenting cells for T‐cell proliferation, they are a poor source of pro‐inflammatory cytokines (Wlodarczyk et al, 2014) and that they differ from infiltrating DC and CD11c− microglia with respect to expression of many genes (Wlodarczyk et al, 2015).