INS and metabolic dysfunction-associated steatotic liver disease: Although the mechanisms involved in the pathogenesis of NAFLD are still not fully understood, the most important theory explaining the physiopathology of NAFLD is the “multiple hit” hypothesis that considers multiple strikes/hits acting together on genetically predisposed subjects such as insulin resistance, hormones secreted from the adipose tissue, gut microbiota, and especially nutritional factors [9,10,11,12].