Finally, it has been recently demonstrated that MALAT1 is overexpressed in mesenchymal stem cells (MSCs) from MM patients and regulates the transcription of the nearby antisense protein-coding gene LTBP3 (latent TGF-β-binding protein) [113], known to positively regulate the activity of TGF-β, which may contribute to the inhibition of terminal osteoblastogenesis in MM [114]. Here, LTBP3 is linked to Miyoshi myopathy.